Tamim Bashir seriously ill

[chinaman]

Tamim Bashir, a promising left-arm spinner from Khulna, has been admitted to a city hospital in a critical condition after he contracted a worst kind of malaria during his stay at the BKSP with the high-performance unit of the Bangladesh Cricket Board.

The disease has affected his brain, blood and kidneys seriously. He is now undergoing treatment at the medicine ward of the Red Crescent Holy Family Hospital. Bashir, who took 44 first class wickets and scored 593 runs, was one of the successful players for GrameenPhone in the Corporate League. His performance helped him earn a place in the high-performance unit which is currently stationed at the BKSP.

NewAge >>

[Optimist]

Very sad indeed. Wishing him a quick and complete recovery!

[chinaman]

Profile: Tamim Bashir

Highlights:

1st Class Batting Ave: 37.06 from 15 matches.

1st Class Bowling Ave: 24.52 from 423 overs with 94 maidens. Total 44 wickets from 1079 runs.

Banglacricket.com prays for his speedy and uncomplicated recovery.

[Piranha]

Praying for a speedy recovery...

If he had Malaria, there is a possibilty that he might have spread it through mosquitoes. I hope this is the last we hear of malaria.

[Zobair]

Very sad news! Our prayers are with him. Inshallah he will recover soon and be stronger than he was before.

[al Furqaan]

let us pray for bashir indeed. only allah can cure him.

[200SX]

we shud pray for him..... if ne1 find ne current news about him plz post it quickly........

[golam]

Lets all pray for his soul now. He passed away.

[al Furqaan]

he passed away??? i can't believe that the medical system is so horrible (wait its bangladesh), never mind.

anyways, innallillahi wa inna ilayhi rajiuun (to allah we belong, and to allah we will return)

[Nasif]

This is extremely sad. We pray for his soul.

It is devastating for a family to loose someone at such a young age. Our sympathy goes to his family. It is a very big loss for all of us.

[chinaman]

May Allah forgive him.

The Daily Janakantha reported the news today.

[billah]

He was a bright cricketer. Very sad indeed. Our prayers are with the family. May Allah accept him in paradise.

[rassel]

Very depressing news indeed! I am very interested to know what exactly happen to him.

[abhs]

Here are some reports

A sad day for cricket-Daily Star,

Cricketer Tamim Bashir dies of Malaria-New age,

Young cricketer Bashir's life in danger-Daily Star,

Tamim Bashirer Okal Mrittu-Prothom Alo.

Mrittur Kase Har Manlen Tamim Bashir-Jugantor (19-06-2004)

Okale ii chole gelen Tamim- Janakantha (19-06-2004)

[Edited on 19-6-2004 by abhs]

[rassel]

Thanks for the link abhs! CEREBRAL MALARIA is a treatable disease! I really don�t to what happen! Anyhow, what can you expect from BD doctor!


here is little info CEREBRAL MALARIA=

What is cerebral malaria?

Cerebral malaria (CM) collectively involves the clinical manifestations of Plasmodium falciparum malaria that induce changes in mental status and coma. It is an acute, widespread disease of the brain which is accompanied by fever. The mortality ratio is between 25-50%. If a person is not treated, CM is fatal in 24-72 hours. The histopathological hallmark of this encephalopathy is the sequestration of cerebral capillaries and venules with parasitized red blood cells (PRBCs) and non-PRBCs (NPRBCs). Ring-like lesions in the brain are major characteristics. Disease risk factors include being a child under 10 years of age and living in malaria-endemic area.

There is a clear need for a strict definition of cerebral malaria in order to properly diagnose and assess the condition. A pragmatic definition based on the Glasgow Coma Score exists. Its key elements are: (1) unrousable coma--no localizing response to pain persisting for more than six hours if the patient has experienced a generalized convulsion; (2) asexual forms of P. falciparum found in blood; and (3) exclusion of other causes of encephalopathy, i.e. viral or bacterial. (Newton and Warrell)

The Blantyre Coma Scale, a related diagnostic tool, has been devised for young children.



What are the symptoms?

Clinical manifestations of cerebral malaria are numerous, but there are three primary symptoms generally common to both adults and children: (1) impaired consciousness with non-specific fever; (2) generalized convulsions and neurological sequelae; and (3) coma that persists for 24-72 hours, initially rousable and then unrousable.



What is the cause?



Sequestration of parasites and obstruction of brain vessels (RPH)

The cause of cerebral malaria is not well understood. Currently, there are two major hypotheses explaining its etiology. They are the mechanical and the humoral hypotheses.

The mechanical hypothesis asserts that a specific interaction between a P. falciparum erythrocyte membrane protein (PfEMP-1) and ligands on endothelial cells, such as ICAM-1 or E-selectin, reduces microvascular blood flow and induces hypoxia. This selective cytoadherence of PRBCs and non-PRBCs, also known as rosetting, can apparently better account for CM�s histopathological hallmark and its characteristic coma condition. However, this hypothesis is inadequate in explaining the relative absence of neurological deficit even after days of unconsciousness.

The humoral hypothesis suggests that a malarial toxin may be released that stimulates macrophages to release TNF-a and other cytokines such as IL-1. The cytokines themselves are not harmful, but they may induce additional and uncontrolled production of nitric oxide. Nitric oxide would diffuse through the blood-brain barrier and impose similar changes on synaptic function as do general anesthetics and high concentrations of ethanol, leading to a state of reduced consciousness. The biochemical nature of this interaction would explain the reversibility of coma.



What are possible treatment options?



As cerebral malaria is the fatal within days of malaria infection if left untreated, immediate treatment is crucial. Because natural immunity to malaria is not fully understood (Immunity) and thus cannot yet be artificially imitated by drugs, control and prevention strategies are significant. Two of these are antimalarial chemotherapy and adjunctive measures. Public health interventions are also critical (Public Health).

P. falciparum parasites in brain capillary (J.S. Tatz)





Chemotherapy for cerebral malaria now primarily involves the use of quinine, for a patient with severe CM must be assumed to have chloroquine resistance. It is one of the four main alkaloids found in the bark of the Cinchona tree and is the only drug which over a long period of time has remained largely effective for treating the disease. Quinine has similar activity to chloroquine in that it is likely to interfere with the parasite�s enzymatic digestion.
Artemisinins have been shown in some clinical trials to clear parasitemia and fever faster than quinine or chloroquine, but they had no effect on mortality rates. Artemisinin has been used by the Chinese as a traditional treatment for fever and malaria. It is a sesquiterpene lactone derived from Artemisia annua. The two most widely used are artesunate and artemether. Because it is both cheap and effective, it is beginning to be included in treatment schedules. However, it is not yet licensed for use in Australia, North America or Europe. Its main value is in the treatment of multi-drug resistant falciparum malaria. As the possibility of quinine resistance looms, artemisinin and its derivatives may soon become the drugs of choice for CM treatment (Newton and Warrell).
Adjunctive measures for CM treatment exist, but they are debatable in both use and efficacy:
Anti-pyretics
Such as paracetamol to reduce fever. However, it is not clear if a reduction in core temperature benefits cerebral consequences.
Anti-convulsants
Such as phenobarbital sodium for seizures. It is crucial to control or prevent seizures, as they can cause neuronal damage and are associated with a fatal outcome.
Reduce intracranial pressure
Using agents such as osmotic diuretics.
Hypoglycemia correction
Using hypertonic glucose. However, theoretically, correcting hypoclycemia in the presence of tissue hypoxia can worsen tissue acidosis.


Exchange transfusion
Generally only been justified when peripheral parasitemia exceeds 10% of circulating erythrocytes. The role of these blood transfusions remains highly controversial, as they are both expensive and potentially dangerous in many malaria-endemic areas.
Anti-Inflammatories
Such as corticosteroids. However, there have been few controlled studies demonstrating benefit.
Desferrioxamine
An iron-chelating adjuvant agent with antimalarial properties. Reduces formation of reactive oxygen species by reducing amount of free iron.
Microcirculatory Flow �
Such as pentoxifylline. Reduces red cell deformability and blood viscosity, decreases systemic vascular resistance, and impairs platelet aggregation, thus improving microcirculatory flow.

(Newton and Krishna). MD



[Edited on 19-6-2004 by rassel]

[AsifTheManRahman]

I was shocked when I read the news in the papers today. This is a very very sad news. May Allah bless his soul.

[mzia]

May all mighty Allah forgive him in all and give the strength to his beloved family members to bear the shock.

The only Allah knows well.

[SS]

deepest condolence to his family and others and wish his soul rest in peace.

[reverse_swing]

Totally unbelievable.May Almighty ALLAH forgive him.

[rassel]

A sad day for Bangladesh cricket

Wisden Cricinfo staff

June 20, 2004



Bangladesh cricket has lost a promising young cricketer. Tamim Bashir, a left-arm spinner from Khulna, died of cerebral malaria at a city clinic on Friday evening. He was 19.


Bashir, who had been training under the guidance of the Bangladesh Cricket Board's high performance unit, had been admitted to the Red Crescent Holy Family Hospital in a critical condition on June 12.


His father Farid Ahmed believes that the indifference of the coaching staff of the high performance unit contributed to his son's death. "My son repeatedly told them that he was not feeling well last Wednesday. But they ignored his pleas and told him to carry on training," Farid told the Daily Star in Bangladesh after the burial of his son.

However, this was refuted by the coach of the high performance unit, who told the paper: "I saw him batting, bowling and fielding on Wednesday (June 9). He looked normal to me. I only got to know about his sickness through his brother on Friday and I told him to stay away from any physical workouts before being recovered.

"It's a tragic loss and the circumstances under which he died is really pathetic. I sympathise with the family and his teammates. If we had known we could have referred him to a BKSP doctor. But by the time we realised Bashir was critically ill he was in hospital."

Bashir was a regular player in the Khulna divisional team. He took 44 wickets and scored 593 runs in the first-class cricket for the side. He was also equally impressive in the recently-concluded Corporate Cricket League playing for the semi-finalists GrameenPhone.

His performance in the national cricket league and the corporate cricket league lead to him earning a place in the Bangladesh Cricket Board's high performance unit.



� Wisden Cricinfo Ltd



[Edited on 20-6-2004 by rassel]

[Huda]

whats with the smile?, u put cric info in right?

it is a sad day, (innalillahi), may allah forgive his sins.

[rassel]

easy sunny boy! That was for cricinfo recognizing tamim!:duh:

[Zobair]

May Allah rest his soul in peace, and give his family the strength to overcome this tragedy. My heart goes out to the parents for whom nothing could be worse than to lose a good son at such an early age in such an abrupt manner.

[babubangla]

While browsing a recent forum thread, I noticed this
thread on Tamim Bashir in the related thread section.
June 18th was his second deah anniversary.

Remembering Tamim Bashir

Cricket lost a bright and promising player with the tragic death of Tamim Bashir on June 18, 2004. The 19-year-old Khulna off-spinner's life came to an end at a city hospital. Tamim had been admitted to the hospital unconscious with cerebral malaria and it was believed the young cricketer contracted the malaria while on vacation in Rangamati. It was too late when he was admitted to the hospital as soon after the admission his kidneys and brain had stopped functioning.
The untimely death of the cricketer had brought a sharp criticism over the then coaching staff of the Bangladesh Cricket Board's High Performance Unit for allowing him to train despite having a fever. In his short career, he appeared in 15 first-class matches and had been a loyal player for Dhanmondi Club in the Dhaka League.

Source: DS

[Sorry]

I lived in rangamati for 2 years and I know how dangerous the mosquitoes are there. Many people take anti-malarial tablets when they visit rangamati.
I pray for the peace in rest.