While we wait for anyone who may be intrested in investigating the above patient (totally understandable if you don't) here's a case people here may be familiar with but I'll put it up all the same
Amoebas are one-celled protozoa. There are several varieties found in humans that are not considered to be disease producers. However, such virulent strains as Entamoeba coli and Entamoeba hartmanni, can produce mild diarrhea and dysentery. Most amoeba infestations, however, do not produce clinical symptoms.
Amoebas generally have a two-phase life cycle: the infective dormant cyst and trophozoite, a later form that is motile and active. When cysts are ingested, they are carried to the small intestine, where they are released as trophozoites into the colon. This form dwells mainly inside the bowel lumen, where it grows and multiplies. The incubation period varies from a few days tothree months. Changes in the host's immune system, or in the organism's pathogenicity, can lead to tissue invasion. The trophozoite can then penetrate through the intestinal lining and invade the liver, lungs, brain, and heart. Subclinical symptoms include the following: upper-right quadrant pain, cramps, occasional nausea, and loose stools. In more serious cases, pronounced abdominal distention, dysentery, fever, and hepatitis may result. Extreme infection can cause abscesses in the liver, the lungs, and the brain.
Chronic diarrhea, gas, and massive food and environmental allergies have all been reported when amoebas are found in the system. Amoebic hepatitis can be mistaken for viral hepatitis; genital amoebiasis for carcinoma; amoebic colitis for ulcerative colitis; and amoebiasis in the brain for a brain tumor. Only a few cysts are needed to cause infection. Amoebic cysts resist iodine and chlorine if concentration of these chemicals is too low.
Two other amoebas responsible for human infection are from the genus Naegleria, which live in freshwater lakes, natural warm water springs, or streams, and can produce encephalitis in swimmers. Although rare, the disease is often fatal. The protozoan Naegleria fowleri is often found in natural warm water springs. It causes a very rare form of meningitis. The amoeba is inhaled and burrows inside the nose, travelling to the brain. Once there, fatal meningitis progresses rapidly. Bathing in the Roman Baths in the city of Bath is no longer permitted because of this protozoa, that has contaminated the water source.
Acanthamoeba species live in soil, as well as fresh and stagnant water, but can be found anywhere. Infections often come as a result of contact wearers not cleaning their lenses with proper solutions, but rinsing them off with tap water. This contamination can lead to eye infections, especially of the cornea, resulting in reduced vision or the removal of the eye (enucleation).The amoeba is also responsible for a severe eye infection called Acanthamoeba keratitis, which results in pain and inflammation around the cornea. If the disease progresses to an ulcer, a corneal transplant is often required. Since relatively few cases are reported, it is assumed that, this too, has been misdiagnosed as another condition.
Endolimax nana is a relatively new member of the pathogenic group of amoebas. It is the smallest of the intestinal amoebas, causing researchers of the past to overlook its potential virulence. This amoeba lives in the lower bowel, but the larvae can sluggishly travel to other parts of the body. It has been linked as a possible cause of rheumatoid arthritis, as well as a host of other collagen-related diseases. (See The Causation of Rheumatoid Disease and Many Human Cancers: A New Concept in Medicine, by Roger Wyburn-Mason, MD, PhD). Typically, other researchers disagree and are looking for another cause.
Entamoeba histolytica is the cause of amoebic dysentery after being transmitted in cyst form from fecally contaminated food or water by way of food handlers (usually aymptomatic carriers), flies, cockroaches, etc. and from certain sexual practices. The disease (amoebiasis) produces abdominal pain and cramps and diarrhea, containing blood, pus, and mucous. A milder form of the disease can display alternating diarrhea and constipation. This disease affects more than 400 million people worldwide, causing mortality second only to malaria. The infective cyst stage develops in the small intestine into the trophozoite stage, where it grows and multiplies in the open spaces of the bowel, feeding on bacteria, tissues, and blood cells. Trophozoites readily die once outside the body, but, inside, they release an enzyme that dissolves tissue, allowing them to penetrate into the intestinal mucosa, where lesions develop and can turn into extensive ulcerative areas that cause dysentery with watery stools containing blood. If the disease disseminates to various internal organs, abscesses usually develop on the liver and possibly on the brain, lungs, heart, or other tissues, and death can result. Most cases are of a mild diarrheal nature or no symptoms at all